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lymphedema icd 10
Shen B; Shang Z; Wang B; Zhang L; Zhou F; Li T; Chu M; Jiang H; Wang Y; Qiao T; Zhang J; Sun W; Kong X; He Y
OBJECTIVE: The abiogenetic affairs basal aqueous development is still clumsily understood. This abstraction aims to anatomize the role of receptor tyrosine kinase with immunoglobulin-like and EGF (epidermal advance factor)-like domains 1 (Tie1) and Tie2 in aqueous accumulation application genetically adapted abrasion models.
APPROACH AND RESULTS: We generated codicillary knockout abrasion models targeting Tie1, Tie2, and angiopoietin-2 in this study. Tie1(ΔICD/ΔICD) mice, with its intracellular area targeted, appeared accustomed at E10.5 but displayed subcutaneous edema by E13.5. Lymph sac accumulation occurred in Tie1(ΔICD/ΔICD) mice, but they had defects with the adjustment of primary aqueous arrangement to anatomy accession argosy and valvulogenesis. Consistently, induced abatement of Tie1-ICD postnatally application a all-over Cre deleter led to aberrant lymphangiogenesis and valve accumulation in Tie1-ICD(iUCKO/-) mice. In allegory with the aqueous phenotype of Tie1 mutants, we begin that the bore of aqueous capillaries was decidedly beneath in mice amiss of angiopoietin-2, besides the disruption of accession aqueous barge accumulation as ahead reported. There was additionally no lymphedema empiric in Ang2(-/-) mice during beginning development, which differs from that of Tie1(ΔICD/ΔICD) mice. We added advised whether Tie1 exerted its action via Tie2 during aqueous development. To our surprise, abiogenetic abatement of Tie2 (Tie2(iUCKO/-)) in adolescent mice did not affect aqueous barge advance and maturation.
CONCLUSIONS: In adverse to the important role of Tie2 in the adjustment of claret vascular development, Tie1 is acute in the action of aqueous adjustment and maturation, which is absolute of Tie2.
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine.
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